This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Aggressive Non-hodgkin Lymphoma
Information about Aggressive Non-hodgkin Lymphoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Aggressive Non-hodgkin Lymphoma
Most recent studies have shown that Aggressive Non-hodgkin Lymphoma shares some biological mechanisms with acute-lymphocytic-leukemia, b-cell-lymphomas, burkitt-lymphoma, chronic-lymphocytic-leukemia, diffuse-large-b-cell-lymphoma, hiv-infections, hodgkin-disease, indolent-non-hodgkin-lymphoma, leukemia, lymphoid-leukemia, lymphoma, lymphoma-follicular, lymphoma-non-hodgkin, malignant-lymphoma-lymphocytic-intermediate-differentiation-diffuse, malignant-neoplasms, malignant-paraganglionic-neoplasm, neoplasm-recurrence-local, neoplasms, t-cell-lymphoma.
Among the many pathways, these few ones have gauged particular interests from scientists studying Aggressive Non-hodgkin Lymphoma, and have been seen in publications frequently: Angiogenesis, Antibody-dependent Cellular Cytotoxicity, Cell Adhesion, Cell Cycle, Cell Division, Cell Growth, Cell Proliferation, Coagulation, Dna Repair, Drug Resistance, Excretion, Immune Response, Interphase, Localization, Metaphase, Methylation, Oncogenesis, Pathogenesis, Programmed Cell Death, Translation
Quite a number of genes have been found to play important roles in Aggressive Non-hodgkin Lymphoma, such as BCL2, BCL6, CSF2, CSF3, DDIT3, DES, EFS, HTT, IL10, IL2, KRT20, MID1, MS4A1, MYC, PGR, TMEM37, TP53. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.